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廣州歐邊生物制品有限公司>>免疫組化>>一抗>>AAT抗胰蛋白酶(兔多克隆抗體)

AAT抗胰蛋白酶(兔多克隆抗體)

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更新時間:2017-12-11 15:24:00瀏覽次數:634

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供貨周期 現貨    
AAT抗胰蛋白酶(兔多克隆抗體)α-1-Antitrypsin 我司為大家提供各種生物原料免疫組化產品,歡迎大家隨時咨詢。

詳細介紹

AAT抗胰蛋白酶(兔多克隆抗體)

α-1-Antitrypsin 

廣州健侖生物科技有限公司

AAT是一種存在于正常人血清中的糖蛋白,可以標記組織細胞與網狀細胞。AAT的陽性染色可用來檢測惡性纖維組織細胞瘤,也可用于篩查隱源性肝硬化和其它不明原因的門靜脈纖維化的肝臟疾病。免疫組化學檢測AAT還可用于檢測遺傳性AAT缺陷,良、惡性肝臟腫瘤,卵黃癌等。

我司還提供其它進口或國產試劑盒:登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

歡迎咨詢

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【產品介紹】

細胞定位:細胞漿

適用組織:石蠟/冰凍

陽性對照:扁桃體

抗原修復:熱修復(EDTA)

抗體孵育時間:30-60min

產品編號產品名稱克隆型別
OB001AACT(抗胰糜蛋白酶)polyclonal
OB002AAT(抗胰蛋白酶)polyclonal
OB003ACTH(促腎上腺皮質激素)polyclonal
OB004Actin,Muscle Specific(肌肉特異性肌動蛋白)HHF35
OB005Actin,Smooth Muscle(平滑肌肌動蛋白)1A4
OB006AFP(甲胎蛋白)polyclonal

 

AAT抗胰蛋白酶(兔多克隆抗體)

阿米巴類感染是由根足蟲綱(Rhizopoda)、阿米巴目(Amoebina)、內阿米巴科(Ent—amoebidae)、內阿米巴屬(Entamoeba)下各種內阿米巴所引起的,臨床習慣簡稱阿米巴。其中腸道阿米巴原蟲,種類雖多,大多寄生于人體內作為共居生物而無致病能力,唯有溶組織內阿米巴寄生于人體后,在一定條件下,可引起疾病,被認為是有致病力的阿米巴。
病原學
溶組織內阿米巴有滋養體及包囊兩期。滋養體自包囊逸出后寄生于大腸腸腔或腸壁,以大腸內容物包括細菌為養料,借腸內乏氧和存在細菌的條件,進行分裂繁殖。滋養體大小不一,12~60um,而以15~30um為常見。
滋養體抵抗力甚弱,在室溫下數小時內死亡,遇稀鹽酸則在數分鐘內死亡。滋養體在適當條件下能侵襲與破壞組織,造成結腸病變,引起臨床癥狀,所以滋養體是溶組織內阿米巴的侵襲型,但它無感染能力。因為在體外它很快死亡,即使進入消化道也很快被胃酸破環。包囊抵抗外界能力很強,在大便中能存活2周以上,在水中能存活5周,能耐受常用化學消毒劑的作用。但對熱和干燥較敏感,加熱至50℃幾分鐘即死。包囊可隨糞便排到外界。人于吞食被包囊污染的食物或水即造成感染。所以溶組織內阿米巴的感染型是包囊。包囊被吞食后,不受胃酸破環,經胃達回腸。由于小腸堿性消化液的作用及蟲體的活動,含有四核的蟲體從囊壁逸出。蟲體又經一系列的復雜變化后,分裂為四個至八個小滋養體,定居于盲腸和大腸近端。
發病原因
包囊被吞食后進入小腸下段,滋養體脫囊逸出,隨糞便下降,寄居于盲腸、結腸、直腸等部營共居生活,以腸腔內細菌及淺表上皮細胞為食。在適宜的條件下,滋養體侵襲腸粘膜,造成潰瘍,到一定范圍和程度時,釀成痢疾。
流行病學
本病見于*各地,其感染率的高低是同各地環境衛生和居民營養狀況等關系極大。溶組織內阿米巴病在熱帶、亞熱帶、溫帶地區,發病較多,以秋季為多,夏季次之。發病率農村高于城市,男子多于女子,成年多于兒童,幼兒患者很少,可能與吞食含包囊食物機會的多少有關。

我司還提供其它進口或國產試劑盒:登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

想了解更多的產品及服務請掃描下方二維碼:

【公司名稱】 廣州健侖生物科技有限公司
【市場部】    楊永漢

【】 
【騰訊  】 
【公司地址】 廣州清華科技園創新基地番禺石樓鎮創啟路63號二期2幢101-103室

The amoebiasis is caused by a variety of endoenterprises such as Rhizopoda, Amoebina, Ent-amoebidae and Entamoeba Cause, clinical practice referred to as amoeba. Including intestinal amoeba protozoa, although many species, most of the parasites in the human body as living organisms without pathogenic capacity, the only solution Entamoeba histolytica parasitic on the human body, under certain conditions, can cause disease, is considered Is a virulent amoeba.
Etiology
Entamoeba histolytica trophozoites and cysts two. Trophoblast escaping from the cyst after the parasitic intestinal or intestinal wall of the large intestine, including the contents of the large intestine, including bacteria as nutrients, by intestinal hypoxia and the presence of bacteria in the conditions of division and reproduction. Trophoblast sizes, 12 ~ 60um, and 15 ~ 30um as common.
Trophozoites resistance is very weak, died within a few hours at room temperature, the case of dilute hydrochloric acid died within a few minutes. Trophoblast invasion and destruction under appropriate conditions in the organization, causing colonic lesions, causing clinical symptoms, so trophozoites Entamoeba histolytica invasion, but it has no ability to infection. Because it quickly dies in vitro, it quickly breaks down even when it enters the digestive tract. Encapsulated resistance to the outside world is very strong, can survive in the stool for more than 2 weeks, can survive in water for 5 weeks, can tolerate the role of common chemical disinfectants. But more sensitive to heat and dry, heated to 50 ℃ a few minutes to die. Encapsulation can be excreted with the outside world. People who ingest swallowed contaminated food or water cause infection. So Entamoeba histolytica infection is cysts. Cysts are swallowed, not by the acid broken ring, up to the stomach by the ileum. As a result of the role of small intestinal alkaline digestive juice and the activity of parasites, tetrazoite-containing parasites escape from the capsule wall. After a series of complex changes in parasites, split into four to eight small trophozoites, settled in the cecum and the proximal colon.
Causes
Encapsulated into the lower intestine after being swallowed, nourish the body to escape, with the drop in feces, colonization in the cecum, colon, rectum and other camps living together to intestinal bacteria and superficial epithelial cells for food. Under suitable conditions, trophozoites attack the intestinal mucosa, causing ulcers, to a certain extent and extent, resulting in diarrhea.
Epidemiology
The disease seen in all parts of the world, the level of infection is highly related to the health status of all localities and residents with great nutritional status. Tissue Entamoeba histolytica in tropical, subtropical, temperate regions, more incidence, with more fall, summer followed. Morbidity is higher in rural areas than in urban areas, with more males than females, more adulthood than children, and very few toddlers, which may be related to the chance of swallowed cysts containing food.

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